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| The long term goals of our research are to unravel root and proximal mechanisms by which endometriosis decreases fecundity. By understanding these mechanisms, novel, targeted approaches for restoration of fertility may be developed. Such approaches are paramount to shift from surgical or chemical obliteration of lesions or repeated attempts at IVF. Comparing data from a rat model to data from women will facilitate mechanistic studies as to how endometriosis reduces fecundity, provide an avenue to test safety and efficacy of new therapeutic approaches, and assist rapid of translation of data into knowledge of human endometriosis. | ||||||||||
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